Anti-Estrogenic Diet/ Hormonal Balancing. In certain problems such as fibrocystic disease, premenstrual syndrome, fibroids, ovarian cysts and some others, an. Zepp Wellness healing, naturally. Anti-Estrogenic Diet. The liver is the primary organ that breaks down estrogen, the hormone responsible for the symptoms in. Anti-Estrogenic Diet. In certain problems such as fibrocystic breast disease, premenstrual syndrome, and some others, an imbalance in estrogen, progesterone.
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Beneficial Conclusion Chapter 3 Exercise Chapter 5 Anti-Estrogenic Foods . Incorporate all anti-estrogenic foods 1–2 fertile eggs (1 yolk) Whey protein .. by this guy named John Collins □□□ anesi.info The Anti-Estrogenic Eating Plan Outline - Free download as PDF File .pdf), Text File .txt) or read online for free. Defense Nutrition Anti-Estrogenic Diet book. ANTI-ESTROGENIC FOOD RECOMMENDATIONS. Many female problems, including uterine fibroids and ovarian cysts are estrogen dependent. Therefore, it is.
The first phase is a liver detoxification program that emphasizes fresh fruits and vegetables especially cruciferous vegetables such as broccoli , low-fat yogurt, organic eggs, beans, whole grains, wild-caught fish and aged cheese. I feel great! In spite of being on more diets than ever, people in this country are becoming even fatter and less healthy. Popular in Food. SlideShare Explore Search You. It is now known that exercise lowers estrogen levels in men and women.
Published in: Full Name Comment goes here. Are you sure you want to Yes No. Show More. Jairo Guizao. Susan Jones. No Downloads. Views Total views. Actions Shares. Embeds 0 No embeds. No notes for slide. The anti estrogenic diet - how estrogenic foods and chemicals are making you fat and sick - 1. In loving memory of my mom, Rina 3. Rick Osborn made important contributions in the editing and designing of the book.
Special thanks to my literary agent Bill Stranger for his support and professional advice. Last, but not least, I thank my wife Natasha for being my biggest supporter in times when I really needed someone to watch my back. How Soy Isoflavones Affect the Body 5. Phytoestrogens in Food, Spices, and Herbs—Harmful vs. I eat as much as I want. I eat only food that I like. I also enjoy overeating and snacking while relaxing in front of TV late at night.
While most people around me are struggling not to gain weight, I find it difficult to keep my weight from dropping.
Here is why. The diet that I practice incorporates certain nutritional elements that are surprisingly missing in most other diets. I believe that these very elements are what keep me lean and healthy. The purpose of this book is to guide you on how to incorporate these critical nutritional elements into your daily routine. These elements are critical because they can help provide you with the means to defend your life. Whether you are aware of it or not, you are constantly exposed to an assault of chemicals known for causing hormonal imbalance, disorders, and death—chemicals that are found in the air you breathe, products you use, These chemicals act in your body like the female hormone estrogen, causing estrogen disorders in women, feminization and sterilization of men, and some of the deadliest cancers in humankind.
To make matters worse, estrogenic chemicals exist in both synthetic and natural forms. The synthetic forms, called xenoestrogens, are industrial derivatives, whereas the natural forms, called phytoestrogens, naturally occur in certain foods, herbs, and extracts.
The double whammy of being exposed to both synthetic and natural estrogens has an overwhelming effect on the body. This is not a simple question to answer. We are currently facing a major ecological problem. These very chemicals, already known for causing the near extinction of various living species today, are currently threatening our own survival.
Yet, this problem is not addressed by any current dietary program. There are many articles and studies that provide much information about the problem but very little about the solution. The Anti-Estrogenic Diet is about solutions. It incorporates nutritional elements that help counterbalance excess of estrogens. It goes head-to-head against common Statistically, most diets today fail in the long run.
Obviously, something must be wrong with those diets. This book explains why diets fail and exposes dietary myths and fallacies. You will learn how wrong your food choices can be in spite of all good intentions. For that matter, you will realize how some of the most popular health-food items today may actually cause weight gain and disorders, whereas some of the foods that you may have been led to believe are fattening can, in fact, help you become lean and healthy.
The following chapters provide you with practical advice that will very likely appeal to your personal needs, lifestyle, and even ethnic background. You can start practicing the program immediately. Over time, you may be able to improve the way you look and the way you feel.
You may also notice a thinning-down effect in areas that previously resisted fat burning, such as around the belly. By improving your metabolic state, you may be able to reverse existing health conditions. Like all humans, you carry within you awesome survival mechanisms. All you need is the knowledge to trigger them.
Once triggered, they will provide you with all the energy and health that you need to better survive. Take advantage of your innate power.
Nourish yourself properly, live better, and live longer. Introduction Excess Estrogen in the Environment and Food There is too much estrogen in the world today. We have never adapted to such a surplus of estrogen in our food and environment, nor have our genes been programmed for such estrogen dominance.
Over-Feminization of the World As a consequence of this imbalance, there is a process of over-feminization that is being noted around the world. Certain species of marine life are becoming sterile and virtually extinct due to petroleum contamination, plasticizers, and other estrogenic chemicals in the oceans, Men, women, and children today are notably getting fatter and heavier due to the effects of excess estrogen. The rates of estrogen-related disorders and cancer in both sexes have reached almost epidemic proportions.
The Anti-Estrogenic Diet The world today has lost its ecological balance. Ironically, many health-food and weight-loss products, including protein bars and shakes, are made with soy protein and petroleum-based chemical additives that promote estrogen. Something must be done to fight this excess of estrogen. That something requires the incorporation of anti-estrogenic foods and nutrients. It also requires a virtual elimination of estrogen-promoting food and chemicals.
This new concept is literally the core of the Anti-Estrogenic Diet. Such a dietary approach may cause controversy and initial resistance. Regardless, all of us—men and women alike—need to follow an anti-estrogenic diet today in order to better survive.
We require food and nutrients that can help support our hormonal integrity—nutrition that can help counter the already existing dominance of We need food that makes us leaner, stronger, and healthier, food that fits our genes—food that is too often deficient in our diets.
Where a reference is not indicated, you should consult the very thorough survey article by I. Munro and colleagues listed in the reference section. As bold as it may seem to be, the Anti-Estrogenic Diet is the only effective way to provide the body with a nutritional means to fight excess estrogen.
Evidence is accumulating that xenoestrogens can disrupt sexual development and reproductive functions of various living species. Some of these chemicals that have penetrated our rivers and ponds—such as plasticizers and petroleum compounds—have been found to cause severe damaging effects on wildlife marine species. Examples of such effects include a decline in the sperm quality of fish, interference with the sexual development of alligators and turtles, and the feminization of male frogs.
Other feminization signs of male fish were found in the Colorado River in white sucker fish. Researchers at the University of California, Berkeley, found that a Researchers believe that chronic exposure to estrogenic chemicals may lead to the virtual extinction of some wildlife species.
Unfortunately, the same chemicals known for causing feminization, sterilization, and the near extinction of various living species today are also found in some of the most common things—the food we eat and the water we drink. Unfortunately, the same chemicals known for causing feminization, sterilization, and the near extinction of various living species are also found in some of the most common things—the food we eat and the water we drink.
According to Turner, et al. Xenoestrogens are also suspected of causing breast cancer cells and vaginal epithelial cells proliferation Krishnan et al. Xenoestrogens, Castration, and Cancer Chemical There is evidence that estrogenic chemicals cause devastating effects on male potency and capacity to reproduce. Exposure of lab species and wildlife to estrogenic chemicals was found to cause abnormalities in Xenoestrogens have shown the ability to bind to estrogen receptors in different glands in both animals and humans, including the gonads, hypothalamus, and pituitary, thereby interrupting their normal functions.
For that matter, xenoestrogens were found to adversely affect testicular gene expression, and also to cause changes in the hypothalamic-pituitary axis in the brain, which is responsible for the healthy regulation of estrogen.
Estrogen is not a single hormone, but rather a group of steroid hormones and their bioactive metabolites. Unlike with other steroid hormones, estrogen receptors are found both in the nucleus and the plasma membrane of cells. That may explain why xenoestrogens have such a high capacity for mimicking estrogen. For example, the chemical BSA conjugated estradiol is a large compound that is unable to gain entry into the cytosol. Nonetheless, it can still bind to estrogen receptors in the plasma membrane and induce an estrogenic effect.
This chemical, also known as synthetic estrogen, was found to cause decreased testicular androgen production in men. BSA is routinely used in commercially raised livestock to fatten the animals to increase profitability.
BSA is also commonly found in the meat we eat commercial beef, chicken, and pork. Xenoestrogens have shown the ability to induce aromatase activity. Aromatase is the enzyme responsible for synthesis of estrogen from the androgens—male Studies showed that over-expression of the aromatase gene and enhanced estradiol production in mice induced spermatogenesis arrest and decreased serum testosterone levels.
Over time, estrogenic chemicals caused progressive degeneration of testicular tissues and sexual behavioral problems in rodents. Elevated blood estrogen levels are known to increase the risk of testicular cancer in men. The harmful effects of xenoestrogens are also evident in humans. A recent study involving a large cohort of men concluded that exposure to the chemical diethylstilbestrol DES caused testicular cancer and malformation of the genitals.
DES is one out of several estrogenic chemicals used as a pharmaceutical agent. Researchers believe that the current increased incidence of testicular cancer in men is most likely due to fetal exposure to estrogenic chemicals, which interfere with the ability of gonadal steroids to support normal tissue differentiation in utero. Elevated blood-estrogen levels are known to increase the risk of testicular cancer in men.
Recent studies at the University of Texas provided evidence that explains the distinct abilities of xenoestrogens to disrupt reproductive functions even at low concentrations.
Researchers tested various xenoestrogens including the phytoestrogen cumestriol found in soy , organochlorine pesticides and their metabolites endosulfan, dieldrin, and DDE , and detergent byproducts of plastic manufacturing The result showed that these xenoestrogens produced rapid estrogen receptor activation 3—30 minutes after application. Furthermore, the same xenoestrogens were also found to be capable of binding and activating estrogen receptors in the pituitary tumor cell line, which expresses high levels of membrane receptors for estrogen.
The pharmaceutical industry argues that xenoestrogens are generally much weaker than the naturally occurring hormone estrogen and therefore unable to compete and induce estrogenic activity. However, there is enough evidence to show that xenoestrogens can bind to estrogen receptors and produce rapid changes in signaling effects, similar to estradiol, the estrogen produced in the ovaries Chen et al. Researchers at Tufts Medical School in Boston found that a certain chemical detergent involved in plastic manufacturing stimulates breast cancer cell growth.
This cancerous estrogenic compound p-nonylphenol belongs to a group of chemical compounds called phthalates, which are mainly used as plasticizers. These increase plastic flexibility—turning a hard plastic to a flexible plastic—and are often found in A correlation was found between heightened levels of estrogenic chemical residues in mothers and smaller penis size of their male children.
Phthalates are also used in the manufacturing of foamed PVC, and they are commonly found in solvents, perfumes, pesticides, nail polish, adhesives, paint pigments, and lubricants. By , manufacturers were producing about one billion pounds of phthalates each year. Phthalates were first produced in the s and have been produced in large quantities since the s, when PVC was introduced. Studies on rodents involving large amounts of phthalates have shown damage to the liver, the kidneys, the lungs, and developing testes.
A study reported that phthalates may mimic estrogen and cause feminization of baby boys Barrett Phthalates are some of the most dangerous chemicals in our world today. Researchers at the University of Missouri conducted studies in which urine samples from pregnant women in four states were collected.
All the women were found to have substantial levels of phthalate residues in their urine. Extra estrogen causes an increase in the size of estrogen-sensitive fatty tissues such as belly Another study revealed that upon birth of children from mothers with phthalate residues, there was a correlation between heightened levels of these estrogenic chemical residues and smaller penis size of their male children.
The evidence of the chemical-castrating effects of these xenoestrogens is overwhelming, yet plastic remains one of the most common, cost-effective ingredients in many products that we routinely use. The manufacturers of plastics and pharmaceuticals do not favor changes in regulations. Not surprisingly, the studies have been criticized. They asserted that the media overstated the finding in the reports. Xenoestrogens, Weight Obesity, and Syndrome X Gain, Estrogen biosynthesis in humans occurs largely in the adipose tissues, which is where fat is stored.
Adipose tissues produce the aromatase enzyme that synthesizes estrogen from the male androgen hormones. Estrogenic chemicals have shown the ability to bind to estrogen receptors and induce aromatase activity in fatty tissue, which increases estrogen levels and activity. This extra estrogen causes an increase in the size of estrogen-sensitive fatty tissues such as belly fat. The There is a direct correlation between elevated estrogen and disorders involving obesity, blood sugar problems, elevated blood lipids, and high blood pressure.
Obesity often involves disorders in blood sugar and hypertension, known as Syndrome X. For that matter, there is a direct correlation between elevated estrogen and disorders involving obesity, blood sugar problems, elevated blood lipids, and high blood pressure. Researchers at the University of Texas-Galveston suggest that the current epidemic of obesity, diabetes, and hypertension may relate to chronic exposure to estrogenic chemicals.
There are over a hundred thousand registered estrogenic chemicals which are currently used worldwide. Only a few of them are regulated in the United States. These substances promote benign and malignant tumors in men and women, and have shown the capacity to induce devastating sterilizing effects on various wildlife species and human reproductive functions.
They are found in virtually every synthetic product that we use or consume. Commonly Used Estrogenic Chemicals Phytoestrogens, also called plant estrogens, have been largely used to treat estrogen-related disorders and cancer. However, there is emerging evidence that some phytoestrogens may, in fact, do the opposite, and under certain conditions cause negative effects similar to estrogenic chemicals.
Phytoestrogens occur naturally in many plants. They have structural and functional similarities to the human hormone estradiol. The most commonly known phytoestrogens are isoflavones, which are found in a By far, the highest amount of these isoflavones is found in soybeans. Other plant compounds with reported estrogenic properties include lignans, coumestans, and lactones. Isoflavones belong to a large group of polyphenolic compounds called flavonoids. Although many flavonoids are often called phytoestrogens, only a limited number have the potency to bind to estrogen receptors and promote estrogenic activity.
Flavonoids often occur in mixtures of estrogen promoters and estrogen inhibitors. Some flavonoids such as chrysin passion flower and apigenine chamomile have shown the ability to inhibit estrogen activity, whereas others such as the soy isoflavone genistein were found to promote estrogen activity.
Flavonoids are known to have widely diverse beneficial biological effects, such as anti-inflammatory, antioxidant, antiviral, and anti-cancerous. Nonetheless, some flavonoids also modulate the functions and metabolism of sex hormones. It is this last property of flavonoids that can either improve or devastate the hormonal balance in the human body.
In times when overwhelming amounts of estrogenic chemicals are penetrating our environment in products, food, and water, it is critically important to know the difference between estrogen promoters and estrogen inhibitors.
There are currently large varieties of commercial products containing phytoestrogens that claim to help treat hormonal imbalance, estrogen disorders, menopausal symptoms, and cancer. The most popular of these are soy products and soy isoflavones supplements. Most studies on soy are sponsored by grants from soy product manufacturers. Consequently, large parts of the current database that we find on soy are positive about the beneficial properties of soybeans.
However, even though less advertised, there are also studies that provide substantial evidence about the harmful effects of soy and its related isoflavones. Soy is a cheap commodity that generates multibillion-dollar profits to soy product manufacturers worldwide. Facing the current popularity of soy as a source of protein and its wide application as a medicinal substance, it is very likely that the following information may appear as highly controversial.
Nonetheless, the information is based on real studies that should not be overlooked.
How Soy Isoflavones Affect the Body Soy isoflavones have been a dietary component for certain populations for many centuries. The consumption of soy has been generally considered to be beneficial. Epidemiological studies over the last decades have suggested that soy isoflavones may provide protective effects against breast, endometrial, and prostate cancer.
Soy protein and soy isoflavones are major ingredients in a large variety of health and weight-loss products. However, recent studies raise serious concerns regarding soy. There is growing evidence that soy may not be as beneficial as it initially appeared to be.
In fact, some studies suggest that soy isoflavones may harm the human body in the same manner as do xenoestrogens. There is also evidence that soy isoflavones may further accelerate the already existing harmful effects of xenoestrogens. Some studies suggest that soy isoflavones may harm the human body in the same manner as xenoestrogens. In one recent study, researchers concluded that soy isoflavones should not be used therapeutically to treat estrogen-related disorders and that more studies are needed to determine the real-life consequences of their estrogenic effects on the human body.
However, in spite of the conflicting evidence regarding soy, the same researchers still suggested that soy is safe. Not surprisingly, they also acknowledged that their review was sponsored by grants from the United Soybean Board, an association of soybean growers.
To understand how soy isoflavones affect the body, we first need to learn about their different forms and bioactivity. Most important is the realization that the body recognizes the highly Dietary sources of soybeans generally contain a mixture of three isoflavones: These three isoflavones appear in two forms: Coumestrol also has been identified as a soy isoflavone, though in lower concentrations than the other isoflavones.
Soy isoflavones may accelerate the already existing harmful effects of estrogenic chemicals and, for that matter, may also contribute to estrogen-related disorders and increased risk for cancer. The biological activity of soy isoflavones depends on their chemical form.
Free soy isoflavones aglycones are more readily absorbed than conjugated isoflavones. In order to be absorbed, isoflavones must be first digested and hydrolyzed into free aglycones. In their natural occurring state, isoflavones exist as glycosides, acetylglycosides, or malonyl glycosides.
Following ingestion, the acetyl and malonyl chains are metabolized to genistein and daidzein, which are then hydrolyzed in the large intestines by gut bacteria, resulting in the production of their respective free-form aglucones, which, as noted, are highly absorbable. Now here comes the interesting part: The isoflavones aglycones are then conjugated in the liver to glucuronic acid and sulfate.
Ninety percent of circulating isoflavones in the body are conjugated to glucuronic acid. Nonetheless, this natural process of neutralization in the liver may present additional problems. Glucuronic acid is a final product of a critically important metabolic pathway in the liver—the glucuronic acid pathway, which is responsible for the transport, metabolism, and neutralization of steroid hormones and their derivatives including estrogenic substances.
Glucuronic acid is an expensive commodity in the body, with a limited supply that generally declines due to high metabolic stress, high levels of toxicity, and aging. Apparently, a high intake of soy isoflavones may rob the liver from its pool of glucuronic acid, leaving it vulnerable to the ongoing assault of other environmental and endogenous estrogens.
Thus, soy isoflavones may accelerate the already existing harmful effects of estrogenic chemicals and, for that matter, may also contribute to estrogen-related disorders and increased risk for cancer. Furthermore, the liver, which has a limited capacity to neutralize toxins, can be overstressed by the overwhelming combination of xenoestrogens, Under such conditions of excess estrogen and a stressed liver, one may be prone to suffer from estrogen-related disorders, fat gain, blood sugar disorders, hypertension, cardiovascular disease, and cancer.
Metabolic Profile and Safety of Soy Due to the structural similarity to endogenous estrogen, there has been considerable research on the effects of soy isoflavones on endocrine functions in humans. A direct correlation was found between soy isoflavone intake and increased levels of circulating estradiol. In a controlled diet study on women, Cassidy et al. Subsequent studies found similar effects.
Soy isoflavones were also found to delay peak progesterone concentrations in subjects consuming only 25—28 mg of soy isoflavones per day. Any substance that suppresses progesterone would leave the body vulnerable to excess estrogen. Low progesterone levels Epidemiological evidence from the Takayama Study Japan suggested an inverse association between serum testosterone levels and soy consumption in Japanese men Nagata Other studies showed no effect.
However, all other studies were short-term and therefore inconclusive. Soy and Infant Development The adverse effects of soy isoflavones are even more pronounced in infants. There have been preliminary reports that isoflavones are excreted through breast milk. Irvine et al. In a controlled study at the University of Iowa, researchers found that women who had been exposed to soy-based formula during infancy would be more prone to suffer from a longer duration of menstrual bleeding and discomfort during menstruation, compared to women who were fed cow milk formula during infancy.
More disturbing is the effect of infant soy formula on the thyroid hormone. Deficiency of thyroid hormone is known to produce goiters in humans and has been reported in infants receiving exclusively soy-based diets. Low thyroid in infants is often associated with stunted growth. A study by Divi et al. Another study featured a case report in which a male infant receiving a soy-based formula was diagnosed with congenital hypothyroidism. Soy isoflavones have also demonstrated adverse effects on reproductive function and development in animal studies.
Estrogen-like effects have been observed in mice at high doses of soy isoflavones. In rats, male reproductive development and mating were reported to be adversely affected by exposure to dietary soy isoflavones. Soy and Carcinogenicity There is evidence that soy isoflavones may induce mutagenic and genetic toxicity. Anderson et al. Even though common opinion considers the genotoxic potential of soy isoflavones as negligible, studies suggest the opposite.
Recognizing the mutagenic effects of soy isoflavones, Alfred et al. Studies in animals showed that administration of genistein to mice produced significant increases in uterine weight and abnormal proliferation of the oviduct cells. In one study, it was reported that prolonged consumption of soy-based diets had a stimulatory effect on proliferation of breast epithelium cells of premenopausal women.
The cancer-promoting effects of isoflavones have been also observed in humans. McMichael-Philips et al. In another one-year study, Petrakis et al. Additionally, in vitro studies found stimulation of growth in human breast cancer cells in postmenopausal women. These studies contradict other studies that found no correlation between soy consumption and breast cancer in postmenopausal women.
Nonetheless, due to the conflicting evidence regarding soy isoflavones and how they affect cancer, it is important to present both sides and let individuals make their choices accordingly.
Soy and Hormonal Replacement Therapy Soy isoflavones have been largely used as a natural alternative for hormone replacement therapy HRT. However, there are contradictory reports in this category as well.
Contrary to the above, Quella et al. A conclusion to the effects of soy isoflavones as a replacement for HRT has not yet been reached.
Soy and Cardiovascular Functions There are conflicting reports about the effect of soy isoflavones on the cardiovascular system. Recent studies reported a significant increase in blood urea nitrogen waste in women consuming soy protein, compared with a control diet. Nitrogen waste is known to adversely affect nitric oxide production and therefore impair vasodilation, while increasing levels of free radicals, which can damage blood vessels.
Soy isoflavones have also been regarded as a cholesterol-lowering agent. Nonetheless, studies state that based on the contradictory results of a limited number of studies, confirmation of the effects of soy isoflavones on serum cholesterol cannot yet be made. Soy and Cognitive Functions It is commonly believed that Japanese people are healthier, slower to age, and live longer than American people. The superior health state of Japanese people has been attributed to their high intake of dietary soy.
However, recent studies revealed that the high consumption of soy may actually have adverse effects on them. A cohort study of Seattle, Washington, Japanese-American men and women indicated that consumers of high amounts of tofu had lower cognitive function scores at baseline.
Also, an association between tofu intake in midlife and reduced cognitive functions, as well as structural changes in the brain, was reported in Japanese-American males. There are conflicting reports on the effects of soy intake on cognitive functions. Some reports support the notion that Japanese are less prone to suffer from age-related brain disease. White et al. Summary Due to conflicting reports on the effects of soy isoflavones on humans, more studies are needed to reach conclusive evidence as to the estrogenic mechanisms of soy isoflavones.
Until then, soy and soy isoflavones should not be used therapeutically to treat menopausal symptoms and estrogen-related disorders. Furthermore, due to the current ongoing assault of xenoestrogens in the environment, food, and water, any estrogen-promoting substance, including soy, may contribute to the problem instead of the solution. Beneficial Any substance that promotes estrogen and suppresses progesterone may contribute to the problem of excess estrogen in the body.
A large variety of phytoestrogens besides soy isoflavones are found in food, spices, and herbs. Some of them are common ingredients in traditional herbal remedies, and are also used today as alternatives for treating estrogen disorders, menopausal symptoms, and cancer.
Studies, however, show that not all phytoestrogens are the same. Recent findings reveal that the different phytoestrogens have different effects on estrogen receptors ER. Some show estrogen-promoting effects, whereas others have Virtually all phytoestrogens were found to be either neutral or antagonistic to progesterone.
Overall, certain phytoestrogens may be harmful, whereas others may be beneficial. Estrogen-Promoting Herbs Recent studies at the Cancer Research Division of the California Public Health Foundation in Berkeley found that the most potent and estrogen receptors-binding products were soy milk, licorice, and red clover.
Licorice and red clover both in the leguminosae family also showed potent progesterone-inhibiting properties. The estrogen-promoting and progesterone-suppressing effects of these herbs should raise serious concerns. As noted, any substance that promotes estrogen and suppresses progesterone may contribute to the problem of excess estrogen in the body.
In , there was an incidence where red clover was found to cause some devastating effects on the reproductive functions of herds of merino sheep in Australia. Other herbs that were found to activate estrogen receptors are dong gui, damiana, black cohosh, verbena, and Their active extracts have shown the capacity to bind to estrogen receptors and stimulate cell proliferation. Commercial black cohosh extract has an extensive clinical history in Western Europe for relief of menopausal symptoms, which are commonly associated with estrogen deficiency.
The notable estrogenic effect of black cohosh may help treat menopausal symptoms; however, there are also disturbing reports on the cancer-promoting effects of this herb in women who already suffer from breast cancer.
Other reports indicate liver-damaging effects of black cohosh after long-term use. Anti-estrogenic Herbs Certain herbs have shown the capacity to antagonize estrogen receptors or inhibit estrogen. Researchers have found that certain flavonoids such as green tea polyphenols and quercetin found in onion and garlic have shown anti-estrogenic, anti-proliferative activity when in high concentrations.
Studies also have found that combinations of resveratrol found in red wine and grapes , apigenine found in chamomile , and quercetin demonstrated substantial anti-cancerous effects on tumor growth, but only apigenine and quercetin showed anti-metastatic effects. Since those contain phytoestrogens, researchers implied that their growth inhibitory mechanism of action is not likely to be estrogen regulated.
Some of these herbs have an extensive history of use as herbal therapies for treatment of cancer. Anti-estrogenic Spices Phytoestrogens are found in spices including thyme, oregano, and turmeric.
However, there is substantial evidence that some of these herbs may actually antagonize estrogen receptors, and therefore, could be highly beneficial as estrogen inhibitors and anti-cancerous nutrients. Thyme and oregano have been used since biblical times as spices and remedies.
For that matter, they are highly beneficial in supporting liver detoxification, and thereby help promote neutralization of estrogenic substances. There is no evidence that thyme and oregano induce any estrogenic effect on the body. Turmeric is a spice commonly used in Middle Eastern, Mediterranean, and Indian foods.
Curcumin, the active ingredient in turmeric and curry exhibited multiple suppressive effects on breast carcinoma cells. Studies in Researchers concluded that curcumin isoflavones could also be used to inhibit the estrogenic effects of pesticides and environmental xenoestrogens. Estrogen-Promoting Spices Contrary to oregano, thyme, and turmeric, there are spices that actually promote estrogen, the most notable of which are licorice and hops.
Studies revealed that all kinds of licorice are estrogenic. Hops is an ingredient in beer that gives the drink its unique bitter taste. Anecdotal reports claim that women harvesting hops often begin menstruation within two days of the beginning of the harvest.
Studies have identified phytoestrogens in hops extracts. It is very likely that the typical beer belly may be related to excess estrogenic activity in the body due to binge drinking.
Summary It becomes evidently clear that when the right choices of food, spices, and herbs are made, applications of natural anti-estrogenic compounds may have preventive and Nonetheless, more studies are needed to elucidate the beneficial or harmful effects of phytoestrogens, in particular those that are commonly found in our food, spices, and herbs.
Due to the conflicting evidence regarding the estrogenic effects of phytoestrogens, there is much confusion today as to what is beneficial and what is harmful. It is therefore important to establish a conclusive database of what are estrogen-promoting substances and what are estrogen-inhibiting substances. There is growing evidence that estrogen-promoting phytoestrogens may accelerate the problem, whereas Just to put things in perspective: For that matter, any viable solution for xenoestrogens must incorporate a daily nutritional regiment based on anti-estrogenic foods, spices, herbs, and extracts—all in sufficient amounts.
Estrogenic chemicals invade our bodies like heavy war machines. We require plenty of anti-estrogenic nutrients—soldiers if you will—to effectively combat, neutralize, and render them inactive within the body. This is the law of nature. Each species has its own specific biological needs.
You never see a cat eating cucumbers, nor a rabbit eating meat—but what about humans? People today habitually use artificial sweeteners instead of natural fruits or grain sweeteners; eat imitation The human body has never adapted to such a chemical insult. Food and Survival Scientists believe that our survival on this planet depended on the capacity to adapt to environmental factors and foods that existed over 10, years ago.
This adaptation process is still deeply carved in our genes. According to the thrifty genes theory, we carry today the same genes as our ancestors, the late Paleolithic cavemen, but the world that we live in today is very different from theirs.
The food that we eat now is not the same as the food that humans adapted to millenniums ago. In a recent review in the Journal of Applied Physiology, researchers suggested that the main cause for the current epidemic of obesity, diabetes, cardiovascular disease, and cancer may relate to the failure of current diet to adequately fit our primal genetic makeup.
Our bodies must be nourished with food that actually fits our genes. Men and women alike are being chronically deprived of certain critical nutrients that would have otherwise provided them with the means to better survive, while sustaining a lean, healthy, and functional body. Along with this view, scientists believe that due to the disappearance of these critical nutrients from our diet, certain positive phenotypes properties of genes—responsible for keeping us alive—are constantly inhibited.
This then causes the over-expression of negative phenotypes, which are responsible for obesity, metabolic disorders, and chronic diseases. Apparently, if we are not actively surviving, we are passively dying.
This all leads to one conclusion: For that matter, our bodies must be provided with additional nutrients that help support and modulate the hormonal system, thereby protecting us against metabolic decline and aging.
Our bodies have been genetically Many people also consume a lot of alcohol, which is known for its estrogen-promoting effect. Diets in this category promote a high consumption of commercial meats and dairy, which are often loaded with hormones and chemical preservatives. To make matters worse, low-carb products are often made with soy protein, petroleum-based sweeteners, and sugar alcohol, all of which contribute to excess estrogen in the body.
Statistically, those on a low-carb diet are prone to suffer from a fat-gain rebound, often regaining more weight than they initially lost. This weight fluctuation may relate It supports the functioning of almost every area of the body, including the brain, heart, bones, muscles, and the immune system. However, if a man's estrogen levels are too high, this can cause a number of health problems, including obesity and depression. Many websites and natural health gurus say that certain diets can lower estrogen levels, but there has been little research into these claims.
A handful of studies have suggested that specific foods can raise or lower estrogen levels. However, scant evidence suggests that these foods can address the health effects of high estrogen.
Some research suggests that the naturally occurring estrogens in plants, for example, do not affect levels of the hormone in male bodies. Speak with a doctor before making dietary changes to reduce estrogen. The most healthful diet varies from person to person. Some research suggests that certain foods may be able to diminish the level of estrogen in the body. However, this research is often low-quality or has involved animals rather than humans.
Soy-based products, including edamame and some meat substitutes, are especially rich in plant estrogens. Estrogens that occur in plants are called phytoestrogens. Early research raised concerns that phytoestrogens could raise estrogen levels in the body. However, they may have the opposite effect. Phytoestrogens are weaker than estrogens that the body produces.
When plant estrogens enter the body's cells, they push out the body's own estrogens. In this way, consuming more phytoestrogens could lower a person's estrogen level. Plant estrogens may also reduce the risk of diseases related to estrogen levels, including prostate cancer. Cruciferous vegetables also contain high levels of phytoestrogens. This group of vegetables includes:. Cruciferous vegetables also contain high levels of isoflavones, which are a type of estrogen.
Results of several studies suggest that isoflavones may prevent the body from converting testosterone to estrogen. Certain varieties of mushrooms , including white button and portobello, could raise a person's testosterone levels and lower their estrogen levels. A study found that curcumin may reduce estrogen levels. However, the researchers noted this result in cells outside the body, so they are unsure whether curcumin has the same effect in people.
A study found that large doses of curcumin increased levels of testosterone in rats. Many farms give female animals, especially cows, high doses of estrogen to increase the amount of milk they produce. Eating these products may increase a person's estrogen levels. A person may wish to switch to alternate milks and sources of protein, or buy meat and dairy products from farms that do not give animals estrogen.
Drinking alcohol may raise the level of estrogen in the blood, which could increase the risk of developing breast cancer.
Alcohol may also heighten some effects of low testosterone. For example, alcohol is high in calories , and it can lead to weight gain. Some grains contain a fungus called zearalenone, which can increase the production of estrogen. Researchers in Europe, where the fungus is common, found that 32 percent of more than 5, mixed-cereal samples were contaminated.
At the moment there is no way to ensure that a product is free from zearalenone.
A person looking to avoid it should limit their intake of grains, including barley, wheat, rice, and maize. Legumes, such as lentils, peanuts, and chickpeas, benefit the health in many ways. For example, they contain relatively high amounts of protein, making them a popular meat alternative.
Legumes also contain phytoestrogens. Particularly in high doses, these foods could increase people's estrogen levels. Legumes may support heart health and reduce the risk of metabolic syndrome.
Rather than removing them from the diet, consider eating small servings a few times a week. Anyone concerned about their levels of testosterone or estrogen should see a doctor before trying to augment these levels.
Few researchers have looked into natural ways to reduce estrogen. However, results of some studies suggest that exercise may lower levels of estrogen in some women. A study from , for example, found that regular exercise reduced levels of estrogen and progesterone in women with a high risk of developing breast cancer.
Also, certain human-made products contain chemical compounds called xenoestrogens, which imitate estrogen in the body. Exposure to these chemicals may also increase the risk of cancer and endocrine disorders. Many plastics contain xenoestrogens. Anyone wishing to lower their exposure to these chemicals may prefer to avoid plastic products, including bottles and food storage containers, when possible.
Men who are concerned about the effects of high estrogen may benefit from making healthful lifestyle changes, such as:. Many myths surround the balance of estrogen and testosterone, especially in alternative health and bodybuilding communities. Several diets promise to lower levels of estrogen, but these claims often lack scientific evidence. Men concerned about changes in hormones or symptoms related to aging should not necessarily assume that high estrogen levels are to blame.
See a doctor, who can test for a range of causes. If a person has a hormone imbalance, treatment is often quick and safe. A doctor can also provide personalized diet and lifestyle recommendations. Article last reviewed by Mon 1 October All references are available in the References tab.
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